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Search Results to Leslie Anne Leinwand

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Leinwand, Leslie

One or more keywords matched the following items that are connected to Leinwand, Leslie

Item Type	Name
Academic Article	Cardiac alpha-myosin heavy chains differ in their induction of myocarditis. Identification of pathogenic epitopes.
Academic Article	Yin Yang 1 is increased in human heart failure and represses the activity of the human alpha-myosin heavy chain promoter.
Academic Article	Yin Yang 1 represses alpha-myosin heavy chain gene expression in pathologic cardiac hypertrophy.
Academic Article	Estimate of the abundance of cardiomyopathic mutations in the ?-myosin gene.
Academic Article	Mutations in the beta-myosin rod cause myosin storage myopathy via multiple mechanisms.
Academic Article	A mutation in the beta-myosin rod associated with hypertrophic cardiomyopathy has an unexpected molecular phenotype.
Academic Article	Insights into human beta-cardiac myosin function from single molecule and single cell studies.
Academic Article	Low sequence variation in the gene encoding the human beta-myosin heavy chain.
Academic Article	Ventricular myosin light chain 1 is developmentally regulated and does not change in hypertension.
Academic Article	Identification of functional differences between recombinant human a and ? cardiac myosin motors.
Academic Article	The Ku protein complex interacts with YY1, is up-regulated in human heart failure, and represses alpha myosin heavy-chain gene expression.
Academic Article	Morphological and functional alterations in ventricular myocytes from male transgenic mice with hypertrophic cardiomyopathy.
Academic Article	Thyroid hormone regulates expression of a transfected human alpha-myosin heavy-chain fusion gene in fetal rat heart cells.
Academic Article	Bioinformatics assessment of beta-myosin mutations reveals myosin's high sensitivity to mutations.
Concept	Ventricular Myosins
Academic Article	Myosin motor domains carrying mutations implicated in early or late onset hypertrophic cardiomyopathy have similar properties.
Academic Article	Divergent Molecular Phenotypes in Point Mutations at the Same Residue in Beta-Myosin Heavy Chain Lead to Distinct Cardiomyopathies.

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Ventricular Myosins